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The etiology in type 1 is thought to be a combination of lack of insulin (+/- glucagon and somatostatin), autoimmunity, autonomic neuropathy, and microvascular damage while the latter two contribute to pancreatic exocrine insufficiency in type 2 diabetes it has been suggested that this may explain why pancreatic exocrine insufficiency is more common in patients with type 1 diabetes (109)
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The three genotypes are highly expressed in synapses before the formation of extensive synapses, with neurexin-1 and neurexin-3 widely distributed between neuronal synapses in the cortical plate, and neurexin-2 distributed between neuronal synapses in the cortical wall [66]
What is the difference between E11.40 and E11.42
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